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Shock

Pathophysiology

  • Initial Stage – Compensatory mechanisms (tachycardia, vasoconstriction) maintain perfusion.

  • Progressive Stage – Oxygen delivery fails → Anaerobic metabolism → Lactic acidosis → Cell dysfunction.

  • Irreversible Stage – Multi-organ failure → Death if untreated.

Types of Shock

Types of Shock
Types of Shock

Hypovolemic Shock (Volume Loss):

  • Cause: Severe blood or fluid loss (e.g., hemorrhage, burns, diarrhea, vomiting).

  • Mechanism: Reduced preload (venous return) → ↓ Stroke volume → ↓ Cardiac output.

Cardiogenic Shock (Pump Failure):

  • Cause: Heart dysfunction (e.g., myocardial infarction, arrhythmias, myocarditis).

  • Mechanism: Inability of the heart to pump blood effectively → ↓ Cardiac output.

Distributive Shock (Vasodilation):

  • Septic Shock: Caused by severe infection and systemic inflammation.

  • Anaphylactic Shock: Allergic reaction causing massive histamine release and vasodilation.

  • Neurogenic Shock: Spinal cord or brain injury causing loss of vascular tone.

  • Mechanism: Peripheral vasodilation → ↓ Systemic vascular resistance → Inadequate perfusion.

Obstructive Shock (Blocked Circulation):

  • Cause: Physical obstruction (e.g., pulmonary embolism, cardiac tamponade, tension pneumothorax).

  • Mechanism: Impaired blood flow → ↓ Cardiac output.

Symptoms

General Symptoms:

  • Hypotension, tachycardia, cold/clammy skin (warm in distributive shock), altered mental status, oliguria, weak pulse, rapid breathing.

Specific Symptoms:

  • Hypovolemic: Dry mucous membranes, reduced skin turgor, flat neck veins.

  • Cardiogenic: Chest pain, lung crackles, distended neck veins.

  • Septic: Early fever/flushed skin, later cool/mottled skin.

  • Anaphylactic: Hives, angioedema, stridor, wheezing.

  • Neurogenic: Bradycardia, warm dry skin, paralysis below injury.

Diagnosis

  • Clinical Assessment: Vital signs, skin color, capillary refill.

Laboratory Tests:

  • Elevated lactate, metabolic acidosis (ABG).

  • CBC, electrolytes, renal function.

  • Imaging: Chest X-ray, CT, ultrasound for causes.

  • Hemodynamic Monitoring: CVP, pulmonary artery catheterization.

Treatment

General Management:

  • Airway: Secure airway, intubation if needed.

  • Breathing: Oxygen or ventilation.

  • Circulation: IV fluids, vasopressors.

Specific Treatments:

  • Hypovolemic: IV crystalloids, blood transfusion if hemorrhage.

  • Cardiogenic: Inotropes, diuretics, revascularization (PCI/CABG).

  • Septic: Antibiotics, IV fluids, vasopressors, corticosteroids if refractory.

  • Anaphylactic: IM epinephrine, antihistamines, corticosteroids, airway support.

  • Neurogenic: IV fluids, vasopressors, spinal stabilization.

  • Obstructive: Pericardiocentesis, chest tube, thrombolytics/embolectomy.

Prognosis and Complications

  • Prognosis: Depends on type, cause, and timely intervention. Early treatment improves outcomes.

  • Complications: Multi-organ dysfunction syndrome (MODS), ARDS, renal failure, death if untreated.


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